Duration of less than 48 hours.
The most common setting for acute and potentially life-threatening hyponatraemia is in the intra- and post-operative setting.
There must be a source of electrolyte free water (EFW):
-
Exogenous (intravenous or oral)
-
Endogenous = desalination of intravenous or body fluids
-
Acute postoperative period
-
Cerebral salt wasting
-
Thiazide diuretics in an oedematous patient
-
Giving isotonic saline to a patient with SIADH
At the same time ADH must be present to prevent the excretion of EFW:
-
ADH release in response to physiologic stimuli
-
Low effective circulating volume:
-
ECF volume depletion
-
Blood loss
-
Hypoalbuminaemia
-
Low cardiac output
-
Excessive pain, nausea, vomiting, or anxiety
-
ADH release without a physiologic stimulus
-
CNS or lung lesions
-
Neoplasms and granulomas such as TB
-
Metabolic lesions e.g. acute intermittent porphyria
-
Administration of agents that stimulate ADH release
e.g. DDAVP or oxytocin
-
Drugs that augment or stimulate ADH release e.g.
morphine, antineoplastic agents
-
Drugs that promote the actions of ADH on the kidney by
increasing cyclic AMP levels or its bioactivity
There are two aspects to management:
-
Prevention
-
Treatment